Dr. Louise Horrigan BSC, MSC, PHD
Contact Details
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Biography:
I graduated with a B.Sc. (Hons) degree in Physiology from NUI Galway in 1998, and then completed a M.Sc. Neuropharmacology also at NUI Galway. I worked for a year in the Infection & Immunity Research Laboratory at NUI Maynooth, before returning to Galway to pursue a PhD in Immunopharmacology. I worked as a fixed term lecturer in the Department of Pharmacology for a period of 4 years, and I am currently employed as a University Teacher in Physiology since 2005. |
Peer Reviewed Journals
| Horrigan, LA,Kelly, JP,Connor, TJ (2006) 'Immunomodulatory effects of caffeine: Friend or foe?'. Pharmacology And Therapeutics, :877-892. [DOI] [Details] |
| Horrigan, L.A.; Holohan, C.A.; Murtagh, M.A.; Williams, C.M.; Webster, C.M. (2013) 'Blueberry juice causes potent relaxation of rat aortic rings via the activation of potassium channels and the H2S pathway'. Food & Function, . [DOI] [Details] |
| Horrigan, LA,Kelly, JP,Connor, TJ (2004) 'Caffeine suppresses TNF-alpha production via activation of the cyclic AMP/protein kinase A pathway'. International Immunopharmacology, 4 :1409-1417. [DOI] [Details] |
Conference Publications
| HORRIGAN, L et al. (2003) Caffeine inhibits monocyte and neutrophil chemotaxis at concentrations relevant to normal human consumption PROCEEDINGS OF THE INTERNATIONAL CYTOKINE SOCIETY ANNUAL MEE , pp.49-54 [Details] |
Abstract
| Horrigan, L.A., Kelly, J.P., Connor, T.J. (2005) Caffeine and its major metabolite paraxanthine suppress human lymphocyte function.(Abstract). Abstract [Details] |
| Horrigan, L.A., Diamond, M., Connor, T.J., Kelly, J.P. (2003) Caffeine consumption may inhibit human neutrophil and monocyte chemotaxis. (Abstract). Abstract [Details] |
| Horrigan, L.A., Kelly, J.P., Connor, T.J. (2003) Caffeine and paraxanthine suppress human TNF-a production via a cAMP/protein kinase A-dependent mechanism. (Abstract). Abstract [Details] |
| Horrigan, L.A., Connor, T.J., Kelly, J.P. (2002) The effects of essential fatty acid deficiency and ethyl-eicosapentaenoate supplementation on immunological parameters in the rat. (Abstract). Abstract [Details] |